投稿信coverletter和highlights模板.docx

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投稿信coverletter和highlights模板.docx

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投稿信coverletter和highlights模板.docx

投稿信coverletter和highlights模板

模板1

DearProf.XXXXX,  

   Herewithinenclosedisourpaperforconsiderationtobepublishedon“(Journalname)”.Thefurtherinformationaboutthepaperisinthefollowing:

 

 

  TheTitle:

    XXXXXXXXXXXXXXXXXXXXXXXXXXXXXXX

  TheAuthors:

  XXXXXXXXXX  XXXXXXXXXXXX and XXXXXXXXXX

 

  Theauthorsclaimthatnoneofthematerialinthepaperhasbeenpub

lishedorisunderconsiderationforpublicationelsewhere.

  Iamthecorrespondingauthorandmyaddressandotherinformationisasfollows,

              

              Address:

   DepartmentofXXXXXXXXX,

                          TsinghuaUniversity,

                          Beijing,10084,

                                        E-mail:

     

               Tel:

      86-

 

               Fax:

      86-

 Thankyouverymuchforconsideration!

 

                                                 SincerelyYours,

 

                                                 Dr.XXXXXXXXXXX

模板2

DearXX(Editor):

Hereisourpapersubmittedto"(XXJournal)".

TheTitle:

 XXXX

TheAuthors:

 XXX XXX and XXX

 

Inthispaper,we...

 

Theauthorsclaimthatnoneofthematerialinthepaperhasbeenpublishedorisunderconsiderationforpublicationelsewhere.

 

ThecorrespondingauthorisDr.XXXandhisaddressandotherinformationisasfollowing:

Address:

DepartmentofXXX,XXUniversity,..., 

Tel:

+86-xxx-xxxxxxxx

Fax:

+86-xxx-xxxxxxxx

 

Thankyouverymuchforconsideration!

SincerelyYours,

Dr.xxx

 

模板3 

DearMr.**

Webelievethepapermaybeofparticularinteresttothereadersofyourjournalasit........

1.Theworkdescribedhasnotbeensubmittedelsewhereforpublication,inwholeorinpart,andalltheauthorslistedhaveapprovedthemanuscriptthatisenclosed.

2.IhavereadandhaveabidedbythestatementofethicalstandardsformanuscriptssubmittedtoNeuroscience.

 

kindregards.

Yoursincerely,

通讯作者

DearProf.Gil:

Thisisamanuscriptby**and**entitled“.......”.Itissubmittedtobeconsideredforpublicationasa“...”inyourjournal.Thispaperisnew.Neithertheentirepapernoranypartofitscontenthasbeenpublishedorhasbeenacceptedelsewhere.Itisnotbeingsubmittedtoanyotherjournal.

Correspondenceshouldbeaddressedto**atthefollowingaddress,phoneandfaxnumber,andemailaddress:

...

Thanksverymuchforyourattentiontoourpaper.

Sincerelyyours,**

 

 

投稿信模版四(实例)

 

Case1

DearEditor,

Wewouldliketosubmittheenclosedmanuscriptentitled"GDNFAcutelyModulatesNeuronalExcitabilityandA-typePotassiumChannelsinMidbrainDopaminergicNeurons",whichwewishtobeconsideredforpublicationinNatureNeuroscience.

GDNFhaslongbeenthoughttobeapotentneurotrophicfactorforthesurvivalofmidbraindopaminergicneurons,whicharedegeneratedinParkinson’sdisease.Inthispaper,wereportanunexpected,acuteeffectofGDNFonA-typepotassiumchannels,leadingtoapotentiationofneuronalexcitability,inthedopaminergicneuronsincultureaswellasinadultbrainslices.Further,weshowthatGDNFregulatestheK+channelsthroughamechanismthatinvolvesactivationofMAPkinase.Thus,thisstudyhasrevealed,forthefirsttime,anacutemodulationofionchannelsbyGDNF.OurfindingschallengetheclassicviewofGDNFasalong-termsurvivalfactorformidbraindopaminergicneurons,andsuggestthatthenormalfunctionofGDNFistoregulateneuronalexcitability,andconsequentlydopaminerelease.TheseresultsmayalsohaveimplicationsinthetreatmentofParkinson’sdisease.

Duetoadirectcompetitionandconflictofinterest,werequestthatDrs.XXXofHarvardUniv.,andYYofYaleUniv.notbeconsideredasreviewers.Withthanksforyourconsideration,Iam

Sincerelyyours,

case2

DearEditor,

Wewouldliketosubmittheenclosedmanuscriptentitled"Ca2+-bindingproteinfrequeninmediatesGDNF-inducedpotentiationofCa2+channelsandtransmitterrelease",whichwewishtobeconsideredforpublicationinNeuron.

WebelievethattwoaspectsofthismanuscriptwillmakeitinterestingtogeneralreadersofNeuron.First,wereportthatGDNFhasalong-termregulatoryeffectonneurotransmitterreleaseattheneuromuscularsynapses.Thisprovidesthefirstphysiologicalevidenceforaroleofthisnewfamilyofneurotrophicfactorsinfunctionalsynaptictransmission.Second,weshowthattheGDNFeffectismediatedbyenhancingtheexpressionoftheCa2+-bindingproteinfrequenin.Further,GDNFandfrequeninfacilitatesynaptictransmissionbyenhancingCa2+channelactivity,leadingtoanenhancementofCa2+influx.Thus,thisstudyhasidentified,forthefirsttime,amoleculartargetthatmediatesthelong-term,synapticactionofaneurotrophicfactor.Ourfindingsmayalsohavegeneralimplicationsinthecellbiologyofneurotransmitterrelease.

 Highlights

Highlightsareashortcollectionofbulletpointsthatconveythecorefindingsandprovidereaderswithaquicktextualoverviewofthearticle.Thesethreetofivebulletpointsdescribetheessenceoftheresearch.resultsorconclusions)andhighlightwhatisdistinctiveaboutit.

Highlightswillbedisplayedinonlinesearchresultlists,thecontentslistandintheonlinearticle,butwillnot(yet)appearinthearticlePDFfileorprint.

Authorinstructions:

HighlightsshouldbesubmittedasaseparatefileinEESbyselecting"Highlights"fromthedrop-downlistwhenuploadingfiles.Pleaseadheretothespecificationsbelow.

Specifications:

Include 3to5highlights.

Thereshouldbeamaximumof 85characters,includingspaces,perhighlight.

Onlythe coreresults ofthepapershouldbecovered.

Examples 

  JournalofHealthEconomics,Volume29,Issue4,July2010,524-535 

Highlights

Wemodeltwohospitalswhichhaveregulatedpricesandcompeteonquality.

Weexaminechangesinthelevelofinformationabouthospitalquality.

Increasinginformationwillincreasequalityifhospitalcostsaresimilar.

Increasinginformationwilldecreasequalityifhospitalcostsareverydifferent.

Welfareeffectsdependonex-anteorex-postassumptionsaboutqualityinformation.

  AppliedCatalysisA,General,Volumes411-412,16January2012,7-14 

Highlights

Highly c-axisorientedZnOnanowiresweregrownonglassusingaqueoussolutions.

Thegrowthtemperaturedoesnotexceed95°Cinanystepofthesynthesis.

ThephotocatalyticandwettingpropertieswerestudieduponUVirradiation.

ZnOnanowiresshowsuperiorphotocatalyticactivity.

Wereportareversiblephoto-inducedtransitionfromhydrophobictosuper-hydrophilic.

  BiochimicaetBiophysicaActa(BBA),Bioenergetics,Volume1807,Issue10,October2011,1364-1369 

Highlights

Aconformationaltwo-statemechanismforprotonpumpingcomplexIisproposed.

Themechanismreliesonstabilizationchangesofanionicubiquinoneintermediates.

Electron-transferandprotonationshouldbestrictlycontrolledduringturnover.

ThemechanismexplainsthefullreversibilityofcomplexI.

  LearningandInstruction,Volume21,Issue6,December2011,746-756 

Highlights

Fadingofascriptalonedoesnotfosterdomain-generalstrategyknowledge.

Performanceofthestrategydeclinesduringthefadingofascript.

Monitoringbyapeerkeepsperformanceofthestrategyupduringscriptfading.

Performanceofastrategyafterfadingfostersdomain-generalstrategyknowledge.

Fadingandmonitoringbyapeercombinedfosterdomain-generalstrategyknowledge.

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